"Today, almost half of our population suffers from a degenerative disease that directly causes Heart Failure, Stroke, Type 2 Diabetes, Kidney Failure, Impotence, Obesity, Neuropathy, Retinopathy and a host of other similar symptoms. It is thought to be implicated, but not yet proven to be causal, in ADHD, ADD and some forms of Cancer. Its beginning symptoms are so mild that most who exhibit them do not realize that they are under a sentence of premature death and disability. It affects adults and children of all ages. At the turn of the last century this disease was so rare that many doctors could not correctly identify it. Today this disease forms such an important economic pillar in the medical community that every doctor in the country can easily recognize it at a glance."


In a time of universal deceit, telling the truth is a revolutionary act.
George Orwell

This website is about a disease syndrome. It is a disease syndrome that is so common that all of us either have the disease or we know someone that does have it. It is a primary, if not the primary, economic support for a medical community whose policy level management has absolutely no interest in curing it. This one disease syndrome alone accounted for over 40% of the deaths in the US from all causes in 1995 (Based on data published by American Demographics). This disease syndrome currently constitutes one of our most effective restraints on population growth in the developed nations. At the turn of the twentieth century, this disease syndrome was so rare that it was considered a medical curiosity.
Much of the information presented on this website has been excerpted from our best selling 147 page Special Investigative Report "Insulin: Our Silent Killer." This report was written by a former victim of this disease who discovered what causes it, when and how and why it became epidemic and how to sucessfully reverse it completely, permanently, economically, naturally and quickly in the large majority of cases. The alternative information needed to cure this disease is not available from the orthodox medical community; indeed, there is some evidence that it has been actively suppressed. Much of this needed information is posted on this website. Additional information, including how to order the special report, that provides substantial additional detail, is provided on our more information page.
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Today an epidemic of incredible proportions rages through the country. It directly affects over half the population and incapacitates almost twenty percent of us. Over ten percent us are completely dependent upon synthetic medication and live under constant medical supervision because of this crippling drug dependence.
There are few of us in America who are not affected directly or indirectly by this disease. It has been known since the 1950's as Insulin Resistant Diabetes, Hyperinsulinemia, or Insulin Resistant Hyperinsulinemia. It is known to the medical community by the symptoms that it produces. Some of these symptoms are: Atherosclerosis, Vascular disease, Diabetes type 2, Impotence, Kidney Failure, Heart Failure, Liver Damage, Stroke, Obesity, Neuropathy, Retinopathy and Gangrene to name but just a few. We have separately discussed, each on its own page, the connection of each of these symptoms to the underlying endocrine disorder, Insulin Resistant Hyperinsulinemia.

This disease has so many life threatening symptoms that it influenced an extensive reorganization of the medical disease classification system in 1949. This was the year the medical community defined many of these symptoms into the independent medical specialty diseases that ravage America today. This reorganization was promoted as a means of focusing medical activity more clearly on the underlying disease syndrome. However, in practice it has resulted in the widespread compartmentalization of the medical community into many different competing medical specialties according to the presentation of the differing "proprietary symptom sets" in different patients.

Doctors now are trained to deal only with their proprietary symptom set according to prescribed protocols. The existence of these numerous new aliases for this disease syndrome became a real problem for the scientific community who has a real interest in understanding cause and effect. Their solution to this "problem of too many aliases" is illuminating. It can be found on our History page.

Also, this compartmentalization resulted in the establishment of powerful economic disincentives to the promotion of a cure for the disease as competing medical specialists vied for market share.

This compartmentalization has obscured both the cause of this disease and the incredible scope of the epidemic that it produces. However, none of these newly formed "medical compartments"are trained to deal with the underlying cause of this disease. As all focus on actually curing anything became hopelessly lost, the orthodox medical community prospered as never before in all of its history. Today America has fallen well below the norm even for western developed nations in the quality and quantity of medical care it dispenses. Here in the US, doctor caused death is the third leading cause of death from all causes. However, the American medical community still retains its lead as the wealthiest and most prosperous medical community in the world.

This compartmentalization resulted in the establishment of powerful economic disincentives to the promotion of a cure for the disease as competing medical specialists vied for market share. Clearly the medical reorganization of 1949, while not all benefiting the patient, did indeed benefit the medical community.

The disease itself that underlies and causes many of these symptoms is discussed on our page on Hyperinsulinemia. Early warning indicators often include elevated Cholesterol Levels, elevated Trigylycerides, poor HDL/LDL ratio, High Blood Pressure and overweight problems. Some of these early warning indicators of disease are discussed on our fats page.

This form of Hyperinsulinemia is also thought to be implicated in several forms of Cancer and in the epidemic of Attention Deficit Hyperactivity Disorder (ADHD) raging through our schools today; although with these latter two diseases, the evidence while compelling is not yet completely conclusive. As we develop the evidence for these connections, we will post them on this site.

Symptoms are valuable warning signs that something is wrong and that quick effective attention is required. When they are deliberately suppressed without curative action being undertaken to cure or reverse the disease, the disease will make more rapid progress. Now that this policy of criminal suppression of symptoms and careful avoidance of cure is being widely exposed, it is now possible to look forward to something other than a rapid decline to invalid status and an early painful death.

One would logically think that this disease would be cured by now since it has been increasingly well understood since about 1950. However, as we shall demonstrate on our history page, there are sound economic reasons why the disease can never, even in principle, be cured by existing medical institutions even though the cure is well understood in the research community. For the 50% or so of people who have the disease in its various stages, it is necessary to seek alternative medical treatment or remain sick.

The good news is that although this disease accounts for almost half of the annual death toll from all causes, it is, in most cases, curable, permanently, quickly, economically, completely and often easily and by natural means. That means little or no reliance upon synthetic designer drugs and no ineffective medical treatments for symptoms while causal agents remain untreated.

The bad news is that the orthodox medical community cannot afford to cure this disease. It forms a financial backbone to the entire orthodox medical establishment. Should an effective cure be popularized, the resulting financial impact to the medical business, the drug business, several of our tax free foundations and a large part of our food processing industry would be severe. Many hospitals would close and many doctors, notably heart surgeons, would soon need to find another line of work. Many drug companies and a large part of the food industry would have to either shut down or greatly change their way of doing business. A discussion of these economic forcing functions is included in our history page.

This disease originated as an epidemic in the late 1920's and became the focus of considerable medical attention in the early 1930's. It grew exponentially through the 1940's and 1950's and is today accepted as a ubiquitous piece of America's medical wallpaper. From a per capita incidence of 0.0028% at the turn of the century, it has ballooned to 10% under doctors care; another 10% who should be under doctors care except they are "coping" and don't yet realize that they are diseased; and another 30% that exhibit clear symptoms which they ignore because the disease does not yet interfere with life. In 1995, forty percent of the death certificates in this country listed one or more symptoms of this disease. Although much has been learned about this disease, dating back to the 1930's, including how to easily cure it, it remains the underlying cause of an incredible annual death toll.

A "death certificate shuffle" exists that acts to obscure the cause of death and to camoflage the incredible epidemic that rages among us. The shuffle works like this. Prior to the reorganization of the medical community in 1949 all of the diseases listed above were known to be but symptoms of what was then called Diabetes. After the reorganization these "symptoms" became diseases in their own right. Each group of related symptoms became the province of the specialist that presumably had expertise with that particular symptom set. For example, if the disease had progressed to the point where the patient experienced heart problems, he would be referred to a Heart Specialist. If he died while treated by the the Heart Specialist, his death certificate would say "Heart Failure." If the disease progressed to the point when Kidney function was destroyed before heart problems occured, the death certificate filled out by the Kidney Specialist would say "Kidney failure."

In this way we have defined a "top ten" category of killer diseases. Many of these killer diseases are but differing symptoms of a single underlying disease with numerous confusing alias's. This underlying disease is now known to be caused by specific poisons and inadequacies in our food supply. For those who would like to see more about the origins of this disease we have included a brief history. As we show on our history page, this disease began to become epidemic shortly after poisonous food was introduced.

This underlying disease is a severe unbalance of the endocrine system that destroys our ability to metabolize food. The unbalance results in elevated levels of certain control hormones as the body strives to correct a systemic problem that it cannot correct without the missing essential elements of nutrition.

Although this is a relatively large site with a considerable amount of useful information, we recognize that many who are either affected by this disease or who have the responsibility to treat others may want to study additional information in report form. For this reason we have compiled a Special Report written for the layman, but which includes an extensive list of scientific cites and references. This report includes a specific, non-drug protocol that can reverse the disease fairly quickly and easily for the large majority that suffer from it. It includes a Glycemic index with complete instructions for use and a Bibliography and a Glossary. This special report contains far more information than we can conveniently present on a webpage. Ordering instructions for this Special Report are also included on our more page.

The results compiled on this web site and in our Special Report are the result of a four year study undertaken by this writer who, having the disease, was faced with the need to find a cure for it because his doctor couldn't or wouldn't cure it. This Special Report presents, not only an effective cure protocol for the disease, but helpful information on how to naturally manage its damaging symptoms during the cure process. It also includes the information needed to reverse much, but unfortunately not all, of the collateral damage that this disease causes when it is encouraged to run rampant with ineffective orthodox medical treatment. Our more page contains much additional descriptive information about our special report along with ordering information.


In the 1880's German scientists wondered what was the function of the Pancreas in anatomy. When they removed the Pancreas from a dog, they noted that its blood sugar rose uncontrollably. Thus the disease that produced high blood sugar came to be known as Diabetes and became identified with a nonfunctioning or improperly functioning Pancreas. Later when Insulin was identified as the causal agent in controlling blood sugar, a search was launched to isolate and synthesize Insulin.

In 1922, three Canadian nobel prize winners, Banting, Best and Macleod succeeded in saving the life of a fourteen year old diabetic girl in Toronto General Hospital with injectable Insulin. Eli Lilly was licensed to manufacture this new wonder drug and the medical community basked in the glory of a job well done. For a time Diabetes, as a disease, was controllable if not curable. No one liked the idea that the diabetic patient had to be supplied with Insulin all of his life, but the idea became accepted as better than premature death.

It wasn't until 1933 that rumors about this disease surfaced in a paper presented by Joslyn, Dublin and Marks and printed in the American Journal of Medicine. This paper "Studies on Diabetes Mellitus", discussed a major epidemic of a disease that looks very much like the Diabetes of the early 1920's only it does not respond to the wonder drug, Insulin. Even worse, sometimes Insulin treatment kills the patient. This disease became known as Insulin Resistant Diabetes because it had the symptoms of Diabetes, but did not respond well to Insulin therapy. Treatment of this disease by diet was started during this period.

Diabetes, which had a per capita incidence of 0.0028% at the turn of the century, had by 1933, zoomed 1000% to become a disease faced by many doctors. This disease was destined to go on to affect half of our population and to incapacitate almost 20% by the 1990's.

It wasn't until 1950 that the medical community was able to perform serum Insulin assays. This quickly revealed that the disease wasn't Diabetes, at least not in the accepted classical understanding of Diabetes. This new disease was characterized by sufficient, often excessive Insulin in the blood. The problem was that the Insulin didn't seem to work to reduce blood sugar; it was ineffective. But, since the disease had been known as Diabetes for almost twenty years it was renamed Type II diabetes to distinguish it from the earlier Type 1 diabetes characterized by insufficient insulin production by the pancreas.

In 1949, faced by what appeared to be an uncontrollable epidemic of the so called Insulin Resistant Diabetes, the medical community reorganized itself into the competing medical specialties that we see today. Thus the "Heart Specialist", "Endocrinologist", "Allergist", "Intestinal Disease Specialist", the "Cancer Specialist" and many others started. Of course, all of these symptoms of this new disease became diseases in their own right. Heart failure for example, which had been previously understood often to be but a symptom of Diabetes, now became a disease not directly connected to Diabetes. It became fashionable to think that diabetes "increased" Cardio-vascular risk. The causal role of a failed Blood Sugar Control System (BSCS) in Heart Failure became obscured.

A year later, in 1950, a search was launched for another "wonder drug" to deal with the Type II diabetes problem. The ideal drug would be, like Insulin, effective in remitting obvious adverse symptoms of the disease, but not effective in curing the underlying disease. It would be needed continually for the remaining life of the patient. It would have to be patentable; that is, it could not be a natural medication because these are non-patentable. Like Insulin, it would be economical to manufacture and distribute. Mandatory goverment approvals would be required to stimulate the use by physicians as a prescription drug. Testing required for these approvals would have to be enormously expensive to prevent other, unapproved medications from becoming competitive. If the drug had unexpected side effects, they could always be explained away by the fact that the disease was worse than the side effects. If the patient died, well, we did our best but this is after all a dangerous disease.

Consider; any drug that would really cure the disease would also put the drug manufacturer out of business in short order due to a lack of customers. Also any drug that was a natural agent, that could not be patented, was not only not a suitable drug company investment but had to be suppressed as unacceptable competition. The reason for this is the huge advertising budget for a natural, unpatented product could not be protected from other companies simply selling the product also without incurring any advertising costs. If the natural competing drug actually worked better than the synthetic drug, all the more reason to suppress it by force of law and to jail its proponents as quacks.

So it was important that the drug be patentable and not natural, effective enough to relieve symptoms, but not effective enough to cure the disease. The medical community also benefited from this approach, not only through the prescription monopoly that they enjoyed, but because the strategy produced the repeat business that they also needed in order to prosper economically.

Thus, was implemented the classic medical protocol of "treating the symptoms". By doing this, both the drug company and the doctor could stay in business and the patient, while not being cured, was often relieved of his symptoms. Enough money changed hands to make the American medical establishment the richest in the world. Unfortunately their patients have not been served as well.

It is important to note that prior to this time the medical goal was to cure disease because the patients usually would not accept anything less. After the introduction of Insulin sucessfully re-trained patients not to expect to be cured, the same commercial technique was applied to the new Hypoglycemic agents. Today all of the Hypoglycemic agents marketed to "control" Type II diabetes meet the original commercial criteria that we have listed above.

In 1955 the oral hypoglycemic drugs were introduced that have been with us for almost five decades. Some of these drugs, notably Rezulin, have been known to kill patients; yet, they remain on the market for unexplainable lengths of time with regulatory agency approval.

Today almost half of the people in the country suffer from one or more symptoms of this disease and it has become well known to physicians asType II diabetes, "Insulin Resistant Diabetes, Insulin Resistance, or more rarely Hyperinsulinemia. One wonders why this "stealth disease" has so many aliases.

When research scientists tried to make sense out of these disease aliases they found conventional medical terminology too confusing to allow useful communication among themselves about the disease. How can you discuss a disease that had as many aliases as there are medical specialties? How does one discuss a disease with their peers that has literally dozens, if not hundreds, of major symptoms and no apparent causative mechanism? This alias, or AKA, type communication problem was resolved when the scientific community abandoned all of the convential medical aliases and identified the disease simply as Syndrome X. It is called a syndrome because it has such a huge number of symptoms. The symptom set includes all of the listed diseases on our home page and many more as well. The basic underlying disorder, Syndrome X, is a derangement of the Blood Sugar Control System by poisonous fats and oils. It is exacerbated and complicated by the near universal lack of other essential nutrition that the body needs to cope with the metabolic consequences of these poisons.

Elevated Cholesterol levels, high Triglyceride levels, Hypertension (High Blood Pressure), inability to metabolize fats and oils, all of the symptoms listed on this home page as well as the well known inability to metabolize Carbohydrates are all symptomatic of this disease. According to the American Heart Association, almost 50% of Americans suffer from one or more of these symptoms. We discuss this from a different perspective on our Hyperinsulinemia page.

When I discovered that this disease appeared quite suddenly in the early 1930's, I wanted to know what else had happened then that might be studied as a causal agent for the disease. It turns out there is a causal agent that originated during the 1920's and which modern biochemists have now connected to the extraordinary disease epidemic in which we are involved. This causal agent is the systematic corruption and commercialization of our food supply starting with our fats and oils.

As early as 1901, efforts had been made to manufacture and sell food products by the use of automated factory machinery because of the immense potential profits that were possible. Most of the early efforts failed because people were inherently suspicious of food that wasn't farm fresh and because the technology was poor. Safeway, for example, was reputed to have chosen its name as an assurance to its customers that it would not sell adulterated food. As long as people were prosperous, "suspicious food products" made little headway. Crisco, the artificial shortening, was given away free in 2 1/2 lb cans in an unsucessful effort to influence the wives of the nation to trust and buy the product in preference to lard.

Margarine was introduced and bitterly opposed by the dairy states. With the advent of the depression, Margarine, Crisco and a host of other Refined and Hydrogenated products began to make significant penetration into the food markets of the nation. Support for dairy opposition to Margarine faded during WW II because there wasn't enough butter for the civilian population and the needs of the military. At this point, the dairy industry having lost much support, simply accepted a diluted market share and concentrated on supplying the military. Flax oils and fish oils, which were common in the stores and considered a dietary staple before our nation became diseased, disappeared from the shelf; the last supplier of flax oil to the major distribution chains was Archer Dainiels Midland and they discontinued the product in 1950.

The history of the adulteration of our once clean food supply exactly parallels the rise of the epidemic Hyperinsulinemia that now lies at the root of many, if not most, of the degenerative diseases from which we suffer.

On our Hyperinsulinemia page we discuss much more about the nature of this disease, including effective ways to reverse its progress. More information is available in our Special Report in hardcopy form about the relationship of artificial fats and oils to this disease. Our Special Report also provides additional detail on the food factors that we must guard against and the best way to recognise the food products that cause this disease.


Allen FM, M.D."Diabetes Mellitus.", Encyclopedia Americana, International Edition 1966 Vol 9 54-56
Brown JAC, M.B., B.Chir.,"Pears Medical Encyclopedia, Illustrated", London, England., Rainbird Reference Books Ltd. 1971
Erasmus U, "Fats that heal, Fats that kill.", Burnaby, BC Canada. Alive Books, 1996
Van Nostrands Scientific Encyclopedia, 8th Ed.
Trager J, "The Food Chronology", Henry Holt & Company. NY, NY. 1995
Return to home page

Diabetes type 2

Diabetes is classically diagnosed as a failure of the body to properly metabolize Carbohydrates. Its defining symptom is a high blood Glucose level. Type 1 Diabetes is caused by "insufficient Insulin" production by the pancreas. Type 2 Diabetes, which constitutes about 95% of all the cases, is caused by normal, or sometimes excessive production of "ineffective Insulin". In both types, the blood Glucose level remains elevated. Neither insufficient Insulin nor ineffective Insulin can limit post prandial (after eating) blood sugar to the normal range; in established cases of Type 2 Diabetes, these elevated blood sugar levels often result in chronically elevated Insulin levels.

The ineffective insulin is no different from normal insulin. Its ineffectiveness lies in the failure of our cell population to respond to it not in any biochemical change in the insulin itself. Therefore it is appropriate to note that this disease is a disease that affects almost every cell in our body. The biochemistry of our cellular metabolism is changed from the normal.

The classification of Diabetes as a failure to metabolize Carbohydrates is a traditional classification that originated in the early 19th century when little was known about metabolic diseases or about metabolic processes. Today, with our increased knowledge of metabolic processes, it would appear quite appropriate to define Type 2 Diabetes more fundamentally as a failure of the body to properly metabolize Fats and Oils as well as carbohydrates. This failure results in a loss of effectiveness of Insulin and in the consequent failure to metabolize Carbohydrates. Unfortunately, much medical insight into this matter, except at the research level, remains hampered by its 19th century legacy.

The Type II diabetes and the Hyperinsulinemic symptoms that occur are system wide symptoms of a basic cellular failure to properly metabolize Glucose. Each cell of our body, for reasons which are becoming clearer, find themselves unable to transport Glucose from the blood stream to their interior. The glucose then either remains in the blood stream or is stored as body fat, or otherwise disposed of in the Urine.

Until quite recently it was believed that Insulin molecules bound themselves to Glucose molecules to form a sort of "lock and key" configuration at the cellular Membrane interface. These two molecules together sort of fit the Membrane Receptor and allowed the Glucose to be transported into the cell where it was used for fuel. Many doctors practicing today were taught this concept in medical school. While this theory is descriptive and attractive in its simplicity and while it may be adequate for some purposes, the true picture that is emerging is much more complex and much more revealing.

It appears that when Insulin binds to a Membrane receptor it initiates a complex cascade of biochemical reactions inside the cell. One of these reactions causes Glucose transporters known as GLUT4 molecules to leave their parking area inside the cell and travel to the inside surface of the cell Plasma Membrane. When in the Membrane, they migrate through the Membrane to special areas of the Membrane called Caveolae areas. There, by another series of biochemical reactions they identify and "hook up" with Glucose molecules and transport them into the interior of the cell by a process called endocytosis. Within the cells interior, this Glucose is burned for fuel by the Mitochondria to produce ATP and waste products. The ATP provides energy to power the cellular activity and the waste products are excreted by other metabolic cellular pathways.

These GLUT4 transporters are responsible for transporting glucose from the blood stream into all of our peripheral cells. Of the seven glucose transporters so far identified they have, by far, the greatest ability to quickly reduce our blood borne post prandial glucose. GLUT1 and GLUT3 are glucose transporters that facilitate the transport of glucose from the blood stream across the blood brain barrier to the neuronal cells of the brain. Remember that the brain uses glucose as a primary fuel almost exclusively. GLUT2 facilitates transport of glucose from our liver and intestines into the blood stream; it also regulates insulin release from the pancreatic beta cells. GLUT5 functions in the absorption of Glucose from the intestine into the blood stream and also in the reabsorption of glucose from the kidneys back into systemic circulation when we are in a glucose sparing mode of operation.. Two additional glucose transporters have been identified but not yet characterized as to their function; they are, as you might expect, GLUT6 and GLUT7.

Each of the glucose transporters operate most efficiently at different levels of blood glucose. GLUT4 swiftly reduces very high levels of glucose. GLUT3 operates efficiently at low blood glucose concentrations in order to keep the brain supplied when blood sugar is low. GLUT2, in its regulatory function, has an activity that is linear across a wide range of blood glucose concentrations. It can thus provide an insulin demand signal to the pancreatic beta cells that is proportional to the blood borne concentration of glucose. Since our BSCS functions as a type 0 control system we would expect to find a proportional sensor somewhere and this seems to be it.

Type 2 Diabetics, perhaps because of the high average Insulin levels, typically have only about 1200 Insulin Receptors, or less, per cell Membrane. This is about half of the norm. This appears to be one of the issues involved in the high average Insulin levels often encountered in Type 2 Diabetes.

Many of the molecules involved in these Glucose and Insulin mediated pathways are Lipids, that is they are Fatty Acids. A healthy Plasma Membrane, now known as an active player in the Glucose scenario, contains a complement of Cis type w=3 unsaturated fatty acids. This makes the Membrane relatively fluid and slippery. When these Cis fatty acids are chronically unavailable because of our diet, Trans fatty acids and short and medium chain Saturated Fatty Acids are substituted in the cell Membrane. These substitutions make the cellular Membrane stiffer and more sticky and inhibit the Glucose transport mechanism. The mobility of the GLUT4 transporters is diminished, the interior biochemistry of the cell is changed and the number of Insulin Receptors on the cell surface is reduced. Although there remains much work to be done to fully elucidate all of the steps in these pathways, this clearly marks the beginning of a biochemical explanation for the known epidemiological relationship between fat metabolism and the onset of Type 2 Diabetes.

There exists another phenomena peculiar to Type II Diabetes that operates to keep the blood sugar elevated. It works like this. Remember that normally the liver stores glucose as glycogen and inhibits the release of glucose when glucose stimulated insulin levels are high. You may recall from our earlier discussion that this mechanism keeps our short term, rapidly accessed supply of glucose replenished. In Type II Diabetes the elevated glucose levels do not seem to inhibit glucogenesis in the liver as it does in normal systems. Although all of the evidence is not yet clear, this can quite likely be due to the same impaired cellular glucose transport at the liver similar to that observed in the peripheral cells. Also when the system is stressed, the Catacholamines that are released stimulate additional glucose release by the liver. Sometimes this effect can be noted by observing a fasting blood sugar in the morning that is higher than the blood sugar of the previous evening, several hours after the last meal of the day. Such unexpected over night elevation of the blood sugar cannot be explained by a meal in the middle of the night when there wasn't one.

It is fashionable for pop medical science to blame "trans fats" for the failure of the plasma cell membranes in our 67 or so trillion cells to facilitate the operation of our GLUT 4 transporters to haul glucose into the cell. To the extent that these trans fats are responsible for stiffening the cell membrane and reducing its fluidity when they are used in place of the Cis type w=3 unsaturated acids, trans fats are indeed a culprit. However, if we didn't eat transfats and ate only "good" short and medium chain saturated fats, we would still get Type II diabetes. The body limits its use of either these transfats or saturated fats in cell membrane repair only when Cis w=3 unsaturated fatty acids are not present in the diet. It is only when these Cis w=3 fats are chronically unavailable to the body that Trans fats and Saturated fats appear in excess in the membranes. These important membranes then stiffen and become sticky, and systemic disease, including Type 2 Diabetes, manifests.

All of these Cis w=3 unsaturated fatty acids have been completely removed from our food chain and replaced by their trans isomer counterparts. This was done because Cis w=3 oils require refrigeration and typically have a fairly short shelf life. This makes them incompatible with the economics of modern food distribution. Our convenient grocery store availablity of these Cis type essential fatty acids ended in 1950 when Archer Daniels Midland, the last supplier, decided to stop producing this valuable oil and concentrate on processing flour. Our diet now consists only of saturated fats, transfats and some of the hardier Cis w=6 and w=9 fats. All of the delicate w=3 Cis fats, uniquely needed by our cell membranes, because of their poor room temperature shelf life, have completely disappeared from the local grocery store.

Two of the w=3 Cis fats are called essential because without them our bodies develop chronic disease and because our bodies cannot synthesize them from any other food that we eat. These two are: Linoleic acid (LA) and Alpha Linolenic acid (LNA). A third fatty acid, Arachidonic Acid (AA), was once thought to be essential. However, recently it has been discovered that a healthy body can convert LA into AA, so it is no longer believed to be essential for healthy people.

A major reason for discontinuing the production of the Cis w=3 oils is that they rapidly become rancid when placed in a transparent bottle on a room temperature grocery store shelf. The resulting manufacturing and distribution problems that would ensue would seriously impact the bottom line of profitability. The trans isomer counterpart to the essential fatty acid we need and do not get has a very long shelf life. Trans fatty acids present few manufacturing or distribution problems for the oil makers; this substitution of the trans isomers for the needed Cis type oils causes Hyperinsulinemia and results in the many other symptoms of the disease that are curently killing us. This outrageous fraud is often accompanied by advertising that informs us of the "monounsaturated" or "polyunsaturated" value of these worthless and damaging trans oils. The law does not require the oil sellers to state that their oil is a trans isomer and not the Cis isomer that we desperately need; and, this is their reason for not doing so.

For more information about these poisonous fats and oils, please visit our fats page.

This story started to come to light in 1950 when serum Insulin assays became available to the medical community. It has been actively suppressed since then; the careers of many ethical scientists have been damaged by trying to bring this story to the light of day. Many others have been cowed into politically correct silence. Now the evidence is so overwhelming and the number of people becoming knowlegable about the matter is so large that it cannot remain hidden in industry funded, politically correct ivory towers any longer. For additional information on the effect of these fats and oils, including how to protect ourselves from the damage they are believed to cause, see our page on Hyperinsulinemia .

For those that prefer more information in hard copy form, this is available in our special report. Please visit this page to see if this is something you would like to know more about.


Le Marchand-Brusted Y, et al, "From insulin receptor signalling to GLUT 4 translocation abnormalities in obesity and insulin resistance.", J Recept Signal Transduct es. 1999 Jan-Jul; 19(1-4):217-228
Dresner A, et al, "Effects of free fatty acids on glucose transport and IRS-1 associated phosphatidylinositol 3-kinase activity.", J Clin Invest 1999 Jan;103(2):253-9
Gustavsson J, et al, "Insulin-stimulated glucose uptake involves the transition glucose transporters to a caveolae-rich fraction within the plasma membrane: implications for type 2 diabetes.", Mol Med 1996 May;2(3):367-72
Farese RV, "Phospholipid signalling systems in insulin action.", Am J Med 1988 Nov;28;85(5A):36-43
Katan MB, et al, "Trans fatty acids and their effects on lipoproteins in humans.", Annu Rev Nutr 1995;15:473-493
Hjelte L. et al, "Pancreatic function in the essential fatty acid deficient rat.", Metabolism 1990 Aug;39(8):871-875


Hyperinsulinemia is an endocrine disorder characterized by a failure of our Blood Sugar Control System (BSCS) to work properly. It manifests when Insulin progressively loses its effectiveness in sweeping the blood Glucose from the blood stream into the sixty seven trillion or so cells that constitute our bodies. Insulin levels in the blood rapidly rise to damaging levels and, together with the resulting elevated Glucose levels, account for much of the damage to our Arteries and Vascular system. When Insulin loses its effectiveness this loss is not due to any change in the Insulin produced by the Pancreas. It is due to a change in the cellular metabolism of almost every cell in our body. Although our Insulin has not changed, our cell metabolism has changed. Our cells no longer respond to blood borne Insulin signalling as they should.

Our Blood Sugar Control System works like any type 0, negative feedback system to maintain our blood sugar at a predetermined setpoint. This setpoint is below the threshold where excess Glucose can cause Vascular damage. And the Insulin required to do this is normally below the threashold where it will cause Arterial or Vascular damage. When the Blood Sugar Control System is working right, it automatically, without our concious knowledge, maintains correct blood sugar with a minimum amount of Insulin whether we have just eaten a meal or been fasting and exercising for a week.

When our system starts to exhibit Hyperinsulinemia, our Pancreatic Beta cells simply increase Insulin production and for a time this maintains our ability to swiftly lower post prandial (after eating) blood Glucose. For a time this maintains normal Glucose levels, albeit by the secretion of these abnormally high Insulin levels.

At some point during the progressive loss of effectiveness of Insulin, our Pancreatic Beta cells may no longer produce enough Insulin to manage normal post prandial and fasting Glucose. This may occur because our Pancreas becomes exhausted by trying to maintain abnormally high Insulin levels needed. It may occur because the progressive failure of our cellular metabolism has created a chronic demand for Insulin beyond what even a healthy Pancreas can supply. In either event, when this happens Type 2 diabetes is diagnosed. Of course, Hyperinsulinemia has been around for some while, often for a long while, by the time this diagnosis is made.

The fat and oils connection to Hyperinsulinemia, and thus to all of the diseases mentioned on our home page, clearly parallels the rise of the Hydrogenated and Refined fats and oils business. Although not well known outside of research circles, (for reasons that are probably economic), the connection between artificial fats and oils and the Hyperinsulinemic destruction of vital functions is now well established. Recent advances in the the study of appropriate cellular biochemical pathways have been most revealing.

To stop and reverse the progress of Hyperinsulinemia the following dietary steps are mandatory:

Do not eat any hydrogenated oils or any prepared foods that contain them as ingredients.
Do not eat any unsaturated or refined vegetable oil that comes in a transparent bottle on a room temperature grocery store shelf. Cold pressed olive oil, coconut oil and Sesame seed oil are the only exceptions to this blanket rule.
Do not eat any oil that has been used in deep frying or that has been heated in cooking except butter and coconut oil. Do not eat any deep fried foods at all.
Add one or two tablespoons full of cold pressed flax oil or fish oil to the daily diet. Do not cook with this oil. Keep it refrigerated when not in use.
Supplement the diet with a good vitamin and chelated mineral complex from a reputable manufacturer. If you don't know one, get a recommendation from your local Naturopath or Chiropractor. Be sure to get Chromium, Vanadium, Calcium, Magnesium, and the trace minerals in the mix.
The program outlined above has a track record of reversing Hyperinsulinemia and the Type 2 diabetes that often accompanies it. In our Special Report, Insulin: Our Silent Killer, we are able to expand, explain and elaborate upon this protocol and present useful tricks and techniques to assure its speedy success in the vast majority of cases.

However, there is another problem that must be faced when curing this disease. It is this: during that part of the cure cycle when elevated blood Glucose and Insulin levels are manifest, the body is being slowly destroyed by these agents. This period may last for many weeks or months dependent upon how long the disease has been allowed to run rampant before a cure is attempted. During this period of time it is of the utmost importance to use all measures available to keep blood sugar and Insulin levels as low as possible so as to minimize the damage. The following list of techniques can be of considerable help in doing this:

Select foods with a low Glycemic Index from a glycemic table. Our Special Report contains a Glycemic Index with instructions for use and much more information on this. Do not depend upon food exchanges; they don't work. They are based on the idea that food may be exchanged on the basis of equal calorie content and ignore that fact that, while calories are important, the glycemic value is far more important to the diabetic.
Use fiber in the diet as much as practical. It reduces the "rate effect" of Blood Sugar Control System perturbation and helps keep post prandial Glucose in check. Our Special Report elaborates upon the types of fiber and on how and why fiber works to control blood sugar.
Develop the habit of exercise at least twice a day. Work for twenty minutes or until you begain to perspire. Do not do aerobic exercises if your Arterial system has been compromised. Our Special Report discusses the additional non-Insulin dependent pathway that exercise opens for Glucose disposal.
Investigate the use of herbs such as Gymnema Sylvestre to help keep blood glucose low. We have more on this Herb as well as over thirty additional hypoglycemic herbs in our Special Report.
Test for allergic reactions when you eat by taking your pulse. When you eat a food to which you are allergic, your pulse rate will escalate at least 10 beats per minute. Don't eat any food to which you are allergic. This technology, too, is expanded and elaborated upon in our Special Report.
As a last resort, consult with your doctor regarding the use of Vanadium Sulfate and other oral hypoglycemic synthetic drugs. Be sure that you understand the side effects of these before you commit to using them. Our Special Report, Insulin: Our Silent Killer discusses rather thoroughly the pros and cons of these medications. It is very useful information to have if you need to discuss these issues with your physician.
In the beginning of the recovery program is is best to remove fats and oils, except flax seed oil, or other oil containing a high percentage of W3 fatty acids, from the diet as completely as possible. Later, when the Hyperinsulinemia has started to reverse, it is ok to gradually restore fats and oils, good fats and oils only, to as much as fifteen percent of the calories consumed.

This program will, relatively quickly, reverse Hyperinsulinemia, Type 2 diabetes and some of the other symptomatic diseases caused by Hyperinsulinemia. In my case, my Type 2 diabetes was reversed in 103 days from start to finish. At the start my fasting blood sugar was 368mg/dl. At the end of the program it varied between 75 and 85 mg/dl.

This program will remove much stress from the components of the Blood Sugar Control System; over a period of time they too will be restored to youthful function. This includes the Liver, Pancreas, Adrenals, Thyroid and the interior transport agents in each cell of our body.

This program will slow and in some cases reverse Vascular damage and Gangrenous damage to our extremities. There are faster ways to reverse this sort of damage which is thoroughly discussed in our Special Report.

This program will, over time, revcrse much of the Neuropathic damage to the nervous system.

It will reverse Atherosclerosis too, but may take years to do it. But here too, there are faster and better ways to reverse Atherosclerosis which we cover in our Special Report.

Although this brief discussion of Hyperinsulinemia has been carefully designed to provide the basic information needed to reverse the condition, a great deal more information is available, in our Special Report, for those who prefer to have a reference book handy. The theory here is that more detail may be needed to assure a comfortable understanding than is practical to include in a web page.


Lepsanovic L. et al, "[Hyperinsulinemia as a key factor in the development of many metabolic disorders]. Med Pregl 1997 Nov;50(11-12):469-472
Nagasaki K, et al, "Relationship between hyperinsulinemia and risk factors of atherosclerosis.", Jpn J Med 1986 Aug;25(3):270-277
Sheu WH, et al, "Insulin resistance, glucose intolerance,and hyperinsulinemia. Hypertriglyceridemia vs hyperl emia.", Arterioscler Thromb 1993 Mar;13(3):367-370
Reaven GM, et al, "Diabetic hypertriglyceridemia.", Am J Med Sci 1975 May;269(3):382-389

Fats and oils

Fats and oils are an important part of any well designed dietary plan. A good working understanding of just what they are and how they work is an essential part of any well conceived diet. Fats and oils, certainly as much and perhaps more than any other single dietary component, directly impact our health in profound ways.

The difference between fats and oils is in their melting point. Fats tend to be solids at room temperature; oils tend to be liquid at room temperature. To turn a fat into an oil, merely raise its temperature above its melting point. If the temperature continues to increase beyond the melting point to the point where some smoke becomes evident, the molecular structure of the oils will change and a number of toxic molecular isomers will be produced in the oil. If the oil is allowed to cool or to resolidify, the toxic products will remain. The temperatures where this damage is done to our fats and oils is about half the temperatures reached in the refining and Hydrogenation processes. Thus, these processes routinely destroy all of the nutritional value of our fats and oils. These refined and/or Hydrogenated fats and oils are characterized by an extraordinarily long shelf life; some are virtually unspoilable.

Naturally occurring fats and oils are Triglycerides. Triglycerides consist of three fatty acids bound to a Glycerol backbone. Each fatty acid consists of a Carbon-Hydrogen chain with a Carboxyl group at the end that is attached to the Glycerol molecule. The other end is typically terminated with a Hydrogen bond. Unless changed chemically, by artificial technology, this is the natural form which we find in the fats and oils that are nutritionally useful. The length of the fatty acid chain as well as its configuration and relative degree of saturation determine how the fatty acid will act within our body. Some fatty acids are vitally necessary to life processes; some are poisons.

Fatty acids are also found in other molecules besides Triglycerides. For example Phospholipids have two fatty acids and a Phosphorus molecule attached to the Glycerol backbone. Phospholipids too, play an important role in our cellular health.

Understanding Triglycerides is an important issue that is complicated by a great deal of pseudo science that is specifically designed to confuse and mislead. In addition to the Triglycerides that we eat in the form of fats and oils, we also have Triglycerides formed, within our bodies, from the sugars and starches that we eat. Much of this Triglyceride load is deposited in our adipose (fat) cells when we eat too much fat and sugar and some of us become obese. Some of these Triglycerides are broken down into their fatty acids which are then used in cell repair. When we lack Cis type w=3's in our diet, most of the fatty acid load is either trans-fats or saturated fats; these are used to repair our cell membranes. It is the combined absence of the Cis w=3 fats and oils and the presence of these saturated and trans-fats and other toxic isomers that cause these cellular membranes to become stiff and sticky instead of fluid and slippery. Additional biochemical detail on this cellular membrane issue is discussed on our diabetes page.

The saturated fat Triglycerides circulate in the blood stream before finding a home in our Adipose cells (fat cells). They tend to be sticky instead of slippery and so contribute to the high incidence of Strokes and Atherosclerosis associated with high levels of Triglycerides in the blood. They make the blood viscosity thicker and cause the Platelets to tend to stick together. They are also an essential step in the chain of events that cause obesity.

All dietary fatty acids may be divided into two categories: Saturated and Unsaturated. The Unsaturated fats and oils differ from each other in their configuration and in their degree of unsaturation. Both types of fatty acids are produced by the the animal and by the vegetable kingdoms, although some are predominately found in animal sources and some are predominately found in vegetable sources. Most concentrated vegetable sources are seeds and nuts; most animal sources are animal body fat. Unrefined fish oils are good sources of dietary Cis w=3 fats; unrefined Flax seed oil, Hemp seed oil and several others are good concentrated vegetable sources of Cis w=3 oils.

Saturated fats are characterized by having all of the possible molecular locations for a Hydrogen bond filled. Thus, at the moludular level, there is no molecular difference between a saturated vegetable fat or a saturated animal fat of the same chain length. There also is no molecular difference between a natural and an artificial saturated fat of the same chain length. Configuration is not an issue because when all of the bonds are filled there is only one configuration possible. As the length of the fatty acid chain lengthens the melting point of the fat increases. Thus fats which are solid at room temperature have longer chain lengths than fats which are liquid at room temperatures. Our bodies can readily process short and medium chain fats; but, it processes longer chain fats with greater difficulty.

However, with animal sources, vegetable sources and even with artificially made dietary sources, single individual fat molecules are never found. We must always deal with mixtures of many different fat and oil molecules in the fats and oils that we consume. All naturally occuring fats and oils are mixtures of long and short chain saturated fats and mixtures of mono and poly unsaturated fats of the Cis configuration. Naturally occuring trans-isomers are relatively rare and do not occur in sufficient abundance to create a health hazard. However if fats and oils are refined, heated or Hydrogenated, the mixtures are then made to also include a huge thermodynamic distribution of highly toxic isomers, including the notorious trans-isomer, along with partially destroyed molecular fragments, and other toxic products.

All fats and oils differ from each other in the length of the Carbon-Hydrogen chain; however, unsaturated fats and oils also differ from each other, and from saturated fats, in that they have one or more vacant Hydrogen sites along their chain.These unfilled Hydrogen binding sites give the unsaturated fats and oils a variety of geometries at the molecular level. Some of these geometries, notably the "Cis" geometries that occur naturally in nature and are designed so that our metabolism can readiy handle them, in fact, it needs them. Certain Cis type unsaturated oils, the w=3's, directly constitute an important building block in all of the sixty seven or so trillion cells in our body, and they cannot be obtained by our body except from our food supply. In addition, our enzyme systems use unsaturated fats as building blocks to construct a wide variety of needed biochemicals

Short and medium chain length saturated animal fats are a very nutritious food staple and have been for thousands of years. They provide nine calories per gram and are "good keepers"; in the days before refrigeration, this "keeping" quality was very important. It meant that the fat would not spoil or go rancid easily at room temperature. Our body uses saturated fats as a highly concentrated energy source when carbohydrates not plentiful. Much of the disease we experience today is the result of a failure of our systems to properly and safely metabolize fats and oils. Rather than use them for the highly concentrated energy source that they are, our body uses them in cell repair because the Cis w=3's are not in our diet.This is now identified as a major factor in Hyperinsulinemia.

Cholesterol is a fatty substance that is manufactured by our liver. It is an extremely important building block for many of our vital functions including our brains, eyes, nervous systems and sexual apparatus (both varieties). About 85% of the Cholesterol circulating in our bodies is made by the liver. We have a Cholesterol control mechanism in our bodies that operates to stabilize Cholesterol at the circulating level that we find. Cholesterol is also contained in some of the foods that we eat. If we try to reduce our circulating Cholesterol by excluding high Cholesterol foods from our diet, our liver simply makes more Cholesterol in an attempt to maintain a homeostasis (normal level) of Cholesterol in our blood stream. Controlling circulating Cholesterol through diet is like trying to empty the ocean with a teaspoon; it sounds like a good pop science theory but it is really not very effective.

As we shall see elsewhere in this website and in our special report Insulin: Our Silent Killer the best way to reduce Cholesterol levels to normal is to cure the underlying Hyperinsulinemia. This entails repairing the Automatic Cholesterol Control System which regulates our Cholesterol homeostasis. This repair process requires stabilizing our blood Insulin and Glucose levels and restoring our entire endocrine system to proper balance. This follows automatically when we stop consuming dangerous, damaged fats and oils and restore other needed nutrition to our diet.

Cholesterol, being a fat, does not disolve in the blood stream which is mostly water. In order to be transported around in the blood, it must be carried by a Lipoprotein carrier which has an affinity for water. When it is being carried from the liver to the rest of the body, the Lipoprotein involved is LDL (low density Lipoprotein). When Cholesterol is being carried from the body back to the liver for recycling, the carrier is HDL (high density Lipoprotein). Thus LDL which distributes Cholesterol throughout the body came to be known as the "bad" Cholesterol and HDL which removes it from circulation came to be known as the "good" Cholesterol. Hyperinsulinemia is characterized by a reduction in the HDL fraction and an increase in the LDL fraction. Clearly this sort of phony science that characterizes one essential Lipoprotein as "good" and another as "bad" is the sort that comes from marketing and sales departments; certainly it does not originate in reputable scientific laboratories.

Besides being a most important building block in many of our bodily functions, Cholesterol is one of the important components of the plaque that occludes our arteries. It is for this reason that it has attracted notice. Our diseased state is due to the fact that the normal levels of circulating Cholesterol have been elevated by Hyperinsulinemia. In fact, this elevated level of Cholesterol is often one of the early warning signs that we are becoming Hyperinsulinemic. An appropriate way to reduce Cholesterol is to cure the underlying Hyperinsulinemia.

With the advent of artificial fats and oils and Hydrogenated and Refined products in the 1920's (see history), the Cis type w=3 unsaturated oils started to disappear from our dietary food chain and were replaced by a large number of toxic isomers. These toxic isomers are just different geometries of the unsaturated oil molecules many of which were, before processing, of the CIS type. Long term consumption of some of these toxic isomers, notably the trans-isomer, has been identified with many, if not most, of the chronic disease symptoms discussed on this home page. Of even greater importance, the complete removal of some of the Cis type w=3 oils from our diet has been found to be causal in many of our widespread degenerative diseases including Hyperinsulinemia.

Some of the biochemical effects of these toxic isomers are discussed on the diabetes page.

Much of this came about because of standardized refining processes that were introduced into the oils manufacturing business. The new rapid high temperature extraction techniques, introduced in the 1920's lowered the retail price of oil, gave it a pure pristine appearance when packaged in a transparent bottle, gave it a uniform clarity, gave it an almost uniform taste, and destroyed the Cis w=3 fatty acids that rapidly spoiled at room temperature. It is the high temperatures used in the refining process that ruins even previously good oils. If we find a good oil and refrigerate it, it is still easy to destroy its nutritional qualities when we cook with it by heating it to the point where it smokes. When delicate Cis w=3 oils are heated, either in cooking or refining, the oil undergoes irreversible changes; the Cis configuration is destroyed and many toxic isomers are generated, including the notorious trans-isomer. All of the antioxidents, previously a part of the unrefined oil are destroyed. Much of the oil's original flavor is lost and it tastes like a generic oil.

When cooking with fats and oils it is important to do so in a manner that does not destroy them. Use only butter, Coconut oil and animal fat for cooking. These contain a higher proportion of saturated fat and thus are not destroyed as easily at cooking temperatures. Never consume any deep fried foods; they are all universally soaked with toxic isomers. If you cook with an oil like olive oil, be sure to mix some water with it to prevent the oil from getting too hot. Remember that if the oil starts to smoke it is too hot and it is being destroyed.

To cure Hyperinsulinemia, Type II Diabetes, Syndrome X and many other consequential diseases that stem from poisonous fats and oils, it is important to realize that the chronic ingestion of Refined and Hydrogenated fats and oils is implicated as a causal agent in these diseases. Margarine, artificial shortenings, refined oils and all Hydrogenated edible products are long term toxic to the human metabolism. Any unsaturated fat or oil that does not need constant refrigeration should be considered unedible. Many saturated fats and oils, while also benefiting from refrigeration, do not turn rancid nearly so easily as Cis w=3 type unsaturated fats and oils at room temperature.

An important consideration about these edible oils is a widespread fraudulent advertising technique that enables the oils manufacturer to sell known toxic oils to the unsuspecting public without breaking the law. Many refined vegetable oils are advertised as monounsaturated or as polyunsaturated in order to confuse the purchaser. Indeed, if these oils were the Cis isomer, they would be desirable oils from a health standpoint. However, Cis type w=3 oils are inherently unstable and will go rancid quite rapidly in a transparent bottle on a room temperature grocery store shelf; their shelf life is on the order of ten hours or sometimes less. The trans-isomer of these oils has a much longer room temperature shelf life. There is no law to require the oils manufacturer, or the store, to advise the consumer that these "monounsaturates" and "polyunsaturates" are trans-isomers and other toxic byproducts that result from the destruction of the good edible oils that they think they are getting. Since no law exists to keep their claims honest, oils manufacturers feel free to deceive with dishonest claims that few consumers understand. In some circles this is not considered to be fraud.

In our discussion on Hyperinsulinemia we discuss more about the fat and oil issue and cover in detail ways we can protect ourselves from the consequences of the fraudulent advertising claims with which we are constantly bambarded. We also discuss how to reverse the degenerative process in the event we are invloved with it.

More information is available in our hardcopy Special Report for those who have a compelling interest or who simply wish to know more about the health connection to our dietary fats and oils.


Erasmus U PhD, "Fats that heal Fats that kill", Alive Books, 7436 Frazer Park Drive, Burnaby BC, Canada 1996
Johnston JR PhD, Johnson IM CN, "Flaxseed (Linseed) oil and the power of omega-3"Keats publishing, Inc. New Canaan, Connecticut.
Beck JS, "Biomembranes: Fundamentals in relation to human biology." NY, NY McGraw-Hill 1980
Enig MG, "Trans fatty acids in the food supply: A comprehensive report covering 60 years of research.", Enig Associates, Inc. Silver Springs, MD1993
Okolska G et al, "[Current recommendations concerning the rational use of fats. II. Value of polyunsaturated fatty acids from the n=6 and n=3 groups and general recommendations]. Rocz Panstw Zakl Hig 1989;40(3):178-187

Folsom AR, et al, "Relation between plasma phospholipid saturated fatty acids and hyperinsulinemia.", Metabolism 1996 Feb;45(2):223-228



Atherosclerosis is a disease which, through several mechanisms, causes the arteries to occlude. Arteriosclerosis is a closely related disease with slightly different causative mechanisms. The occlusion that these diseases cause effectively reduces the interior diameter of the Artery. Because of this reduced interior cross section of the Artery, less blood can flow through the Arterial system at normal blood pressure. The body then either increases the blood pressure or we find ourselves frequently "out of breath" as we try to "make do" with a lessened oxygen transport around the body. Eventually, if the Coronary Artery is involved, as it often is, insufficient oxygen delivery to the Heart muscle results in Heart Failure. If the Renal Artery is involved insufficient blood supply to the Kidneys can result in Kidney Failure.

The material that accumulates on the interior walls of the Artery is a plaque-like substance that typically has a calcium substrate that holds it in place. Among the mechanisms thought to be causative in the development of plaque in the Arteries are: Insulin damage to Arterial Endothelium that causes the bodies Arterial repair mechanism to patch the damage with plaque, stimulation of uncontrolled replication of Cytomegalovirus infected Arterial Intima cells by Insulin like growth factor and deposits of Cholesterol on a Calcium substrate in the Artery.

An elevated Insulin level, Hyperinsulinemia, is directly implicated in the damage done to the Arterial Endothelium that stimulates the repair response; it is also responsible for the stimulation of Intima replication and in the depressing of HDL and the elevation of LDL. This change in the HDL/LDL ratio results in higher than normal Serum Cholesterol levels. Some of this excess Cholesterol is thought to constitute the construction material for Arterial Plaque. To cure the Atherosclerosis requires the removal of the plaque from all of the Arteries. To prevent a return of the Atherosclerosis it is necessary to cure the Hyperinsulinemia that is a precipating cause of it. Modern orthodox medical practice does neither.

Conventional orthodox treatment for Atherosclerosis includes Arterial replacement, often referred to as "bypass surgery", and balloon angioplasty. Neither treatment addresses the cause of the disease. Neither treatment fixes the rest of our Arteries which are also occluded, possibly to a less than an immediate life threatening extent. Both treatments are expensive, invasive and very dangerous. Some patients die on the operating table. There are no scientific studies to support the use of these techniques. In addition, only the Artery, or Arteries, that causes the most life threatening symptom is treated. After treatment, the disease in all of our remaing untreated Arteries will continue to progress; often it will continue at at an accelerated rate. These treatments are time consuming, very expensive and very dangerous. The few times any of these treatments have ever been subjected to double blind studies to assess their usefulness they failed to demonstrate any improvement in the long term survival rate at all. See the article on Heart failure.

Successful unorthodox treatment must include reversal of the damage done to the Arteries as well as a reversal of the plaque forming mechanism that caused the problem in the first place. Chelation therapy has been used very successfully for about forty years to remove the plaque that has accumulated in the Arteries, all of the Arteries simultaneously. It is fast, efficient and economic in approximately 80% of the cases; also, risk is virtually non-existent. Detailed information about Chelation therapy can be obtained from our reference #5 listed below. Once the Arterial plaque is removed by Chelation, Hyperinsulinemia, the systemic disease characterized by the high Insulin levels, must be cured or the Arteries will simply begin the occlusion process all over again. See our page on Hyperinsulinemia for more information on this killer; included there is information on how to avoid it and on how to cure it if you haven't successfully avoided it.

It is important to note that Atherosclerosis reduces, sometimes greatly, the delivery of oxygen and other vital nutrients to the sixty seven trillion or so cells that comprise our body. For this reason many diseases of impaired circulation, some of them potentially fatal, are a direct result of Atherosclerosis.

For those who are interested in the source data from which these conclusions are drawn, a starter list of references is included below and much more information is available in our Special Report. There are hundreds of excellent studies available to support the conclusions found in this web page.


Goalstone ML, et al. "Insulin potentiates platelet-derived growth factor action in vascular smooth muscle cells.", Endocrinology 1998 Oct;139(10):4067-4072
Stout RW. "Insulin as a mitogenic factor: role in the pathogenesis of cardiocascular disease.", Am J Med 1991 Feb21;90(2A):62S-65S
Fujii K, et al. "Association between hyperinsulinemia amd intima-media thickness of the carotid artery in normotensive men.", J Hypertens 1997 Feb;15(2):167-172
Agewall S, et al. "Carotid artery wall intima-media thickness is associated with insulin mediated glucose disposal in men at high and low coronary risk.", Stroke 1995 Jun;26(6):956-960
American College for Advancement in Medicine, 23121 Verdugo Drive, suite 204, Laguna Hills, CA 92653 (714) 583-7666 (800) 532-3688 FAX (714) 455-9679

Kidney failure

Technically termed Renal Nephropathy, Kidney Disease is an inevitable result of the chronically uncontrolled blood Glucose and chronic high Insulin levels that are associated with Hyperinsulinemia and Type 2 diabetes.

The Kidneys are system filters whose purpose is to filter out the water soluble waste materials in the blood, to mix them with an appropriate amount of water to form urine and to send this mixture to the bladder for excretion from the body. They do this in a way that conserves blood components that may be needed for future use. Thus the Kidneys return non-waste blood components to the blood stream to maintain proper blood Homeostasis, they control the blood PH to within very tight limits (7.35-7.45), they control the water Electrolyte blood balance and they, through the action of Renin-Angiotensis mechanism and the excretion of water from the system, play an important role in the operation of the Blood Pressure Control System (BPCS). This BPCS is another of the bodies' control systems similar to, but not directly related to, the Blood Sugar Control System.

In structure the Kidneys consist largely of many fine capillaries through which the blood flows during the filtration process. A number of complex osmotic reactions occur during the passage of blood through the Kidneys that are driven by the Electrolyte balance in the blood and by the osmatic pressure differentials caused by the Sodium Potassium balance in the blood stream.

When Renal Failure occurs, Dialysis is required or death will ensue in short order, typically 50% within six months and most by one year. Dialysis is a procedure whereby the blood is externally filtered through a machine especially designed for the purpose. When this procedure is done periodically the blood stream is filtered of impurities. By the use of this method life may be prolonged after Kidney Failure is experienced.

There are three major damage mechanisms by which progressive deterioration of the Kidneys occurs.

The first is by direct damage of the fine capilaries by the high Glucose levels associated with Diabetes and with Hyperinsulinemia. The glucose directly causes an increased permeability and the capillaries leak and fail to perform their filtering action. Severe and extensive damage to these fine capilliaries is thought to be irreversable at this time.

A leading cause of Renal Failure for the Hyperinsulinemic patient is Renal Thrombosis (blood clotting in the Renal vein).

The high levels of Insulin in the blood directly cause Atherosclerosis of the Renal artery. This mechanism is discussed on our Atherosclerosis page.

A related complicating factor is caused by the high levels of dietary protein to which many sufferers from Diabetes and Hyperinsulinemia resort. This protein, when it is metabolized, results in high levels of Ammonia . Chronic high levels of Ammonia, mostly changed into Urea by the Liver, nevertheless directly damage the fine capillaries. Also there are known other negative effects in the tubular basement membranes of the Kidneys that are directly caused by certain proteins.

Unless normal blood Glucose levels can be maintained without resorting to a high protein diet, the progress of Hyperinsulinemia and its related Type II diabetes invariably leads to Renal (Kidney) failure and the need for Dialysis. In our special report Insulin: Our Silent Killer we thoroughly discuss the dietary factors that are especially important to the diabetic. This High Protein-Kidney Damage relationship is potentially one of the most important to the recovering diabetic. During the recovery process, which may last for several months, it isn't smart to wreck the Kidneys by resorting to a high protein diet. This is uniquely important because, while much of the damage done by Type II diabetes and Hyperinsulinemia is reversable, all of it is not. In particular when the basement membranes of the Kidneys are destroyed, the destruction is currently thought to be irreversable.

High blood pressure, Hypertension, which is often found in progressive Renal Failure, is also one of the characteristics of Hyperinsulinemia. If there is any suspicion that Hyperinsulinemia or Type II diabetes, may exist, one should have a blood test done and obtain medical advice based on the BUN and Creatine levels found in the test with regard to possible Kidney damage.

On the next page where we discuss Hyperinsulinemia, we discuss ways in which this disease can be readily prevented from progressing. We include below a starter list of references for those who want to look at the original data. Much more information about Hyperinsulinemia is available in our Special Report than can conveniently be put on this web site.


Burton C et al, "The role of proteinuria in the progression of chronic renal failure.", Am J Kidney Dis 1996 Jun;27(6):765-775
Marieb EN, PhD, "Essentials of Human Anatomy and Physisology", The Benjamin Cummings Publishing Company, Inc. 1997
Haller H, "Postprandial glucose and vascular disease.", Diabet Med 1997 Aug;14Suppl3:S50-S56
Leutenegger M, "[Theoretical aspects of the relationship between diabetic microangiopathy and hyperinsulinism.]", Presse Med 1992 Sept;921(28):1324-1329
Stern MP, "The effect of glycemic control on the incidence of macrovascular complications of type 2 diabetes.", Arch Fam Med 1998 Mar;7(2):155-162

Liver damage

The Liver is a huge chemical factory that manufactures basic biochemicals that are used by our entire body. It is central to our digestion of fats, to metabolism, to detoxification and to many other vital processes. When it fails, death will typically occur within 24 hours.

Detoxification, one of the many vital functions of the Liver, is performed through multiple chemical pathways. These pathways chemically alter toxic materials into harmless substances that are water or fat soluble. The water soluble end products are sent to the Kidneys for excretion in the Urine. The fat soluble end products are sent to the small intestine for subsequent excretion.

Virtually all synthetic drugs, as well as many natural drugs and compounds, are regarded by the Liver as toxic. For this reason many, if not most, prescriptions for synthetic drugs call for up to 50 times the dosage needed to combat the symptom treated. The assumption is made that sufficient drug must be introduced into the system to overcome the detoxification activity of the Liver.

This is one of the reasons why most, if not all, modern synthetic drugs have one or more undesired side effects. Many of the Hypoglycemic agents (agents that lower blood sugar) used to treat Type II diabetes and Hyperinsulinemia are known to cause liver damage. There are so many of these agents, with more appearing every day, in an exploding drug marketplace, that it isn't possible to list and discuss them all on this web site. Some of the toxic effects of these drugs affect other organs of the body in addition to the liver. An increasingly common side effect is that they kill. In our Special Report we thoroughly discuss the common side effects of all of the current classifications of Hypoglycemic agents.

When you contemplate taking a prescripton drug it is very important to understand the consequences of these drugs. You can visit the library to look up the drug in the "Physicians Desk Reference" (PDR). Or, alternatively, if you have our Special Report you can quickly look up the risk factors involved with the medication you're taking in the appropriate section of the report. This will eneble you to determine the nature of its side effects. If you are already experiencing any of these side effects it is time to have a serious talk with your physician. We cite here side effects for a few of the commonly prescribed agents used to control blood sugar in Hyperinsulinemic and Type II diabetic patients so that you can get the idea.

The Sulfonylureas are a class of drugs that include Orinase, Tolinase, Diabinase and others. These drugs have been implicated in greatly increased deaths from Heart Disease and Hepatitis, specifically Granulomatus Hepatitis.

Glyburide, another Hypoglycemic agent has been reportedly associated with Cholestatic Jaundice, with Hepatitis and with Hypersensitivity Angitis; all of these are very serious Liver diseases.

Rezulin is an oral Hypoglycemic agent that has come to be associated with Liver Cancer. It was believed to be a potent Cancer producer by the original FDA investigating officer who refused to approve it for American use. He was administratively removed from the case and replaced by another who quickly approved it. After approval it killed an estimated 100 people before recently being finally removed from the American market by our FDA. It had previously failed to gain regulatory approval with England's equivalent of our FDA. This matter is currently headed for the courts.

In addition to drug induced Liver damage, evidence is slowly accumulating to suggest that the Hyperinsulinemia itself may cause Liver damage. High levels of Triglycerides in the blood stream are a known result produced by the development of Hyperinsulinemia and Hyperglycemia (Type II diabetes). When these Triglyceride levels become chronic, they induce a condition known as Hepatic Steatosis (fatty liver). In this condition, the Liver becomes infiltrated with Triglycerides and much of its normal function is impaired.

In our section on Hyperinsulinemia we present effective ways to deal with this disease. Below we include a starter list of references for those who wish to do further investigation. In addition to the contents of this web page, which have been designed to include the essential information needed, more information is available in our Special Report on this disease.


Saw D, et al, "Granulomatous hepatitis associated with glyburide.", Dig Dis Sci 1996 Feb;41(2):322-325
Wanless IR, Lentz JS," Fatty liver hepatitis (steatohepatitis) and obesity: an autopsy study with analysis of risk factors.", Hepatology 1990 Nov;12(5):1106-1110
Whitaker JM, "Reversing Diabetes.", Warner Books, Inc. 666 Fifth avenue, NY, NY. 10103


Although Impotence is a disease that has a variety of causal agents, some of them not even medical in nature, Hyperinsulinemia is directly implicated in Impotence through at least three biochemical pathways. These pathways are Neuropathy of the affected nerves and ganglia, Atherosclerosis and Vascular insufficiency. All three of these pathways are characteristic of Hyperinsulinemia and the diabetes that often occurs along with it.

Some studies show that up to 35% of males who suffer from diabetes are also functionally Impotent. Of course, Impotence along with its causal agent Hyperinsulinemia, is readily reversible in the vast majority of cases, without the need for synthetic drugs of any kind. The 35% figure undoubtedly provides much of the economic incentive for the recent introduction of the drug Viagra.

Impotence is often the result of the Diabetic Neuropathy that develops as a result of high uncontrolled Glucose levels. Some of the parasympathetic nerves involved include the Pacinian Corpuscles of the Penis, the Pudential nerve that passes to the Dorsal Root Ganglia, the Perivesicular, Prostatic and Cavernous Plexes and the Postganglion fibers that innervate the smooth muscles of the Vas Deferens, Seminal Vesicle and the Internal Sphincter of the Bladder. Any or all of these nerves can and do fail to operate properly when subject to Neuropathic damage from high Glucose levels. See our page on Neuropathy for more details.

The erectile mechanism works by increasing blood flow through the right and left Internal Pudendal Arteries to the right and left Corpora Cavernosa and by decreasing the Venous blood outflow. The high Insulin levels associated with Hyperinsulinemia cause Arterial occlusion and result in an insufficient blood supply to the Corpus Cavernosa. The high Glucose levels associated with this disease results in increased Venous permeability; thus the blood outflow from the organ is not sufficiently restricted. See our page on Vascular disease.

Viagra, a popular new synthetic drug, has been introduced by Pfizer for the specific purpose of improving erectile response in Impotent males. While it is developing a track record of working in some, but not all cases, there are some precautions that should be taken and some risks that should be weighed before embarking upon a program of using this drug. Viagra is known to increase the Hypotensive effects of Organic Nitrates; if you are taking Organic Nitrates in any form it is not smart to use Viagra. Also a serious list of side effects seems to be characteristic of this drug. Some of the most important of them, together with their relative frequency of occurance are:

16% Headache
10% Flushing
7% Dyspepsia
4% Nasal congestion
3% Urinary tract infections
3% Abnormal vision
3% Diarrhea
2% Dizziness
2% Rash
2% Other
The "2% other" in the above list is particularly interesting. Among the "other" numerous side effects can be found the following:

Heart failure
Angina pectoris
Face Edema
Allergic Reaction
Rectal Hemorage
and many others
When these side effects were listed in the PDR (Physicians Desk Reference) it was stated, by the manufacturer, that the causal relationship between Viagra and these adverse reactions has not been clearly established; these reactions simply occurred to volunteers involved in the clinical tests. About 3700 were involved to some extent in the tests with 550 involved for more than a year.

For those whose Atherosclerosis involves the Carotid or Coronary arteries, as it often does, the use of Viagra has the potential of precipitating a fatal Heart Failure event or stroke; and as a result, the drug should not be used by these patients.

By far the best and safest approach to the problem of Hyperinsulinemic caused Impotence is to simply cure the Hyperinsulinemia. This will swiftly arrest the progress of the Impotence. To restore proper functioning in a wholly natural and normal manner it is necessary to clean out the clogged Arteries. We introduce this idea on our page Atherosclerosis. It is thoroughly discussed in our Special Report; see our more information page.

Although Hyperinsulinemia is not the only cause of impotence, there are many others, it is a major cause of this malady.

We provide below a starter list of references. A much more extensive Reference list, together with a Bibliography for further research, can be found in our Special Report, Insulin: Our Silent Killer.To reverse impotence safely, it is necessary to reduce the elevated levels of blood Glucose and Insulin that often are the causative agents and to restore proper operation of our automatic Blood Sugar Control System. Much more information on this subject is also included in our Special Report.


Russel JW, Feldman EL, "Insulin-like growth factor-I prevents apoptosis in sympathetic neurons exposed to high glucose.", Horm Metab Res 1999 Feb-Mar;31(2-3):90-96
Buvat J, et al, "Mechanisms of diabetic impotence. 25 cases.", Presse Med 1987 Apr 11;16(13):611-614
McCulloch DK, et al."The prevalence of diabetic impotence.",Diabetologia 1980 Apr;18(4):279-283
Jevtich MJ, et al, "Vascular factor in erectile failure among diabetics.", Urology 1982 Feb;19(2):163-168


Stroke is defined as the sudden rupture or occlusion by clotting of a blood vessel to or within the brain. Symptoms depend greatly on which blood vessel is affected.. They can range from minor dizziness or momentary temporary loss of function that is barely perceptible to paralysis or death.

Hyperinsulinemia and the high blood Glucose levels that come with it produce two effects that are responsible for inducing Strokes. These effects are: (1) increased thickening of the blood and increased stickiness which tends to clot and block the blood vessels and, (2) the increased permeability of the microvascular system that cause the smaller blood vessels and capilliaries to leak and rupture.

Ischemic strokes are produced by blood clots that may occur at the site or may occur because of a clot that, having been formed elsewhere, was carried to the site by blood flow. When blood becomes thicker and more sticky this type of stroke becomes more likely. The high levels of Glucose and the high levels of Triglycerides that characterize both Type II diabetes and Hyperinsulinemia make the blood sticky, viscous and prone to clotting. This is why these diseases are often associated with Ischemic Stroke.

Hemorrhagic strokes are caused by rupture or severe leaking of the blood vessels. This is usually caused by Glucose induced damage to the blood vessel, particularly when small Veins or Capilaries are involved. Chronic elevated levels of blood Glucose are usually implicated in Hemorrhagic stroke.

The cause of the blood changes that induce both types of Stroke are found to be characteristic of the Hyperinsulinemia syndrome. Below we include a starter list of basic references for those so inclined. More information along with extensive scientific cites and source references are available in our Special Report. This report, Insulin: Our Silent Killer discusses much more fully the Hyperinsulinemia that is so often causally implicated in the incidence of stroke.


Davis TM, et al, "Risk factors for stroke in type 2 diabetes mellitus: United Kingdom Prospective Diabetes Study (UKPDS) 29.", Arch Intern Med 1999 May 24;159(10):109710103
Pyorala M, et al, "Hyperinsulinemia and the risk of stroke in healthy middle-aged men: the 22 year follow-up results of the Helsinki Policemen Study.", Stroke 1998 Sep;29(9):1860-1866
Kamide K et al, "Insulin resistance is related to silent cerebral infarction in patients with essential hypertension.", Am J Hypertens 1997 Nov;10(11):1245-1249
Biessels GJ, "Cerebral complications of diabetes: clinical findings and pathogenic mechanisms.", Neth J Med 1999 Feb;54(2):35-45
Lukovits TG, et al, "Diabetes mellitus and cerebrovascular disease.", Neuroepidemiology 1999;18(1):1-14



When the study for this web site started some years ago, we assumed that the problem of Obesity, as an issue in Hyperinsulinemia and Type II diabetes, was one that was relatively well understood and thoroughly researched. We were surprised to discover that less is known about the weight control mechanism, the hunger reflex and their relationship to Hyperinsulinemia than is known about vitually any other aspect of this dangerous disease.

During our research we found many competing theories with various credibility levels. The best of them, though incomplete, made sense and came from some of the most reputable research labs around the world; the worst of them made no sense at all and typically originated in the sales departments of the diet and weight loss industry.

We present here that which we think makes the most sense and which will likely remain as a basic fundamental to a full understanding of Obesity, if and when it ever becomes available.

With Hyperinsulinemia, the excess average Insulin levels experienced cause an increase of Lipogenesis. Lipogenesis is the Insulin mediated conversion of Glucose into Triglycerides (fats). The resulting high Serum Triglycerides are stored in adipose cells throughout the body; these adipose (fat) cells become distended and as they distend, more Insulin is required to enable them to carry on their metabolic processes. There is but a finite, fixed number of fat cells available in each body. This number is changed only with difficulty as we mature. The excess Triglyceride load that they absorb in Obesity simply makes each adipose cell larger.

As these fat cells absorb the excess Triglyceride load, they emit a biochemical into the blood stream called Leptin. This is a biochemical that causes a reduction in the amount of a Neural Polypeptide called NPY (neural polypeptide Y) which is produced in the Hypothalamus. The Hypothalamus is the part of our brain known to mastermind many of the bodies regulatory mechanisms. The body weight seems to depend upon the ratio of Leptin, emitted by the fat cells, and NPY produced by the Hypothalamus. When there is too much NPY, and possibly other more obscure Neural Peptides, Glucose transfer into the cells is inhibited, Lipogenesis is stimulated and Triglycerides are stored in fat cells. When there is too much Leptin, fat storage is supposed to be inhibited.

This same Hypothalamus also masterminds the control of the Appestat (hunger regulator). The role of NPY and Leptin, if any, in the hunger reflex is not well understood yet. But, clearly there is a connection because the only way to provide the additional Triglyceride load for the fat cells to store is to stimulate the hunger reflex to require more eating of carbohydrates and of fats and oils.

In the case of Obesity, the NPY and Leptin levels remain elevated and it appears that the appropriate Leptin receptors in the Hypothalamus are desensitized. The result is that the Hypothalamus continues to insist upon Triglyceride storage in the adipose cells, by emitting NPY, and it continues to maintain a hunger reflex beyond any reasonable need to do so. We get fat.

One theory, which has some epidemiological support, may be helpful. It says that because the food we eat is so empty of needed nutrients, our hunger reflex remains activated and the role of Leptin remains suppressed. This is done in the body's expectation that, if we continue to eat, eventually the needed nutrient will magically appear in the stomach. The assumption here is that when the needed nutrient appears, the Leptin-NPY axis will again function and we will lose weight. This theory may be fruitful in the effort to more fully understand the connection between the Hypothalamus, which is widely considered to be a lower center control mechanism, and the higher centers of our brain where our intelligence resides.

At any event, while the connection between Obesity and Hyperinsulinemia, is not yet fully understood, much more is known about the cause, progress and cure of Hyperinsulinemia and Type II diabetes.

For those who would like to pursue the study of Obesity beyond these meager beginnings we include a starter reference list. Judicious use of it can quickly get you into the mainstream of what's going on worldwide in this area. More data is included in our Special Report on the Hyperinsulinemia that is so often causal in our Obesity and weight control issues.


Boden G, "Free Fatty Acids, Insulin Resistance, and Type 2 Diabetes Mellitus." Proc Assoc Am Physicians 1999 May9;111(3):241-248
Jeanrenaud B, et al, "[From Claude Bernard to the regulatory system between the hypothalamus and the periphery: implications for homeostasis of body weight and obesity]." C R Seances Soc Biol Fil 1998;192(5):829-841
Stejskal D, et al, "[leptin, insulin and proinsulin--their relationship]." Vnirt Lek 1998 Jun;44(6):361-365
Koyama K, et al, "Tissue triglycerides, insulin resistance, and insulin production: implications for the hyperinsulinemia of obesity." Am J Physiol 1997 Oct;273(4Pt):E708-13
Rohner-Jeanrenaud E, Jeanrenaud B, "Central nervous system and body weight regulation.", Ann Endocrinol (Paris) 1997;58(2):137-142


Neuropathy is a generic medical term for a group of diseases of the nerve. They affect both motor and sensory nerves with varying results. Diabetic Peripheral Neuropathy is a term used to describe damage done to the nerves by high Glucose levels in the blood stream. Generally this high Glucose level causes a deterioration of the Mylin Sheath surrounding the central nerve and leads to its degeneration.

When the affected nerve is a motor nerve, progressive loss of ability to stimulate the related muscle structures causes loss of function.

When the affected nerve is a sensory nerve, two different results may occur. Often only the sensory function is impaired and a gradual deterioration in feeling and sensation occur. Sometimes, when the Mylin Sheath becomes damaged, intense pain results which necessitates the use of narcotic pain killers.

When sensation is essential to function, as for example in the male erectile response, function also becomes impaired. See our page on Impotence.

Sometimes when the damage done to the nerve causes loss of function, as with a Heart muscle, the consequences can result in Heart Failure and death.

All parts of our complex nervous system can be affected by high average Glucose levels; this includes the Central Nervous System, the Voluntary Nervous System and both the Sympathetic and Parasympathetic elements of our Involuntary Nervous System.

When the Blood Sugar Control System is repaired and again functions properly, Neuropathy, along with many other symptoms of impaired Glucose control will reverse. Nerves will regenerate. They will regenerate faster when supplied with the unique nutrients needed to repair cellular and plasma membrane damage.

This Neuropathy is one of several extremely dangerous side effects of a Blood Sugar Control System that does not function to maintain normal levels of both Glucose and Insulin in our bodies. Neuropathy is a normal consequence of Hyperinsulinemia. Below we include a starter list of references for those who like to study the original material. Much more information is included in our Special Report which also offers a methodology that has a track record of reversing the underlying cause of this disease.


Mahgoub MA, Abd-Elfattah AS, "Diabetes mellitus and cardiac function.", Mol Cell Biochem 1998 Mar;180(1-2):59-64
Tutuncu, Bayraktar M, Varli K, "Reversal of defective nerve conduction with vitamin E supplementation in type 2 diabetes: a preliminary study.", Diabetes care 1998 Nov;21(11):1915-1918
Dahl-Jorgensen K, "Diabetic microangiopathy." Acta Paediatr Suppl 1998 Oct;425:31-34
Cameron NE, Cotter MA,"Metabolic and vascular factors in the pathogenesis of diabetic neuropathy.", Diabetes 1997 Sept;46Suppl2:S31-37
Marieb EN, "Essentials of Human Anatomy and Physiology" Fifth edition, The Benjamin/Cummings Publishing Co, Inc. 2725 Sand Hill Road Menlo Park, CA 94205

More information
Hyperinsulinemia is an explosive epidemic that currently affects half of us to a greater or lessor degree. In the middle 1920's this disease, then called Diabetes, was so rare that many doctors never saw it. Some ethnic communities, notably the Chinese, had never had a single case reported. Others, like the American Eskimo, who subsisted on a diet of about 60% saturated fat, had no incidence of this disease at all. Today, half the American population suffers one or more symptoms of this disease.

It is caused by nutritionless food. This food is sold to us for the purpose of making a profit and generating growth in the competitive companies that constitute the processed food industry. Our food lacks essential nutrition because the nutritious substances in it have a very short shelf life. Since this abreviated shelf life is incompatible with modern food merchandising technology, the nutrition is modified or removed. The food is then sold as "fortified", or as "new and improved". The direct result of this lack of nutrition never affects the sale of the food because these consequences are delayed by months or years. By that time the customer (victim?) has either forgotten the bad food or been trained to believe that it is innocuous.

All of us, without exception, have been programmed by the intense advertising and marketing promotions to which we are subjected almost from birth. Much of this stuff is designed to get into our subconscious by bypassing our normal conscious censor mechanisms. Once there, these implanted perceptions take on an emotional value that makes us want to defend them as if they were our own ideas. While there might once have been some legitimate rationale for merchandizing to sell products, it has become dangerously ubiquitous, intense, deceptive and effective. Subliminal advertising practices are widespread in the selling of food products to America. Without constant, informed effort to maintain our objectivity, we easily fall prey to deeply held beliefs placed in us without our consent or knowledge.

Which of us doubts that our food industry really has maintaining our health as their highest priority instead of maximizing their corporate bottom line. Yet this semireligious belief is ridiculous on the face of it. Who among us doubts that the medicines routinely sold to the public have been carefully and honestly cleared by government regulatory agencies. We have no doubts even when our newspapers are full of articles about the death dealing side effects of these drugs. Why do we continue to believe that our regulatory agencies act without being influenced by commercial interests even when we read horror stories like the approval of Aspartame. Why, after drugs like "Rezulin", formulated to treat this disease, has killed well over one hundred trusting people, do we continue to trust the integrity of the drug manufacturer who continued to sell it? Even after knowing that our regulatory agencies permitted Rezulin to remain on the market to kill more long after the lethal nature of the drug was widely reported, how many of us really suspect that anything is wrong? How many of us abandon this nonsense and seek out an alternative non-lethal treatment? It is only when we become directly and personally impacted, as was this writer, that we begin to study carefully what is really going on in this commercial arena.

Such is the power of the subliminal advertising to which we are all exposed every day.

Perhaps some insight may be gained into the scope of the health disaster in which we are involved by noting the the largest processed food manufacturer in the world is Kraft Foods. Kraft Foods is owned by Phillip Morris; yes that's right, its the same Phillip Morris that gave us the tobacco scandals of the late 1990's.

Although we have been careful to supply the needed information that can help to completely reverse Hyperinsulinemia and Type II diabetes in this website, we are aware that some of the information contained here is new for many of us. Sometimes with new information, especially when it may tend to contradict previously held beliefs, more support and evidence is required to bring the new data within our comfort zone.

We have prepared a 147 page Special Report entitled "Insulin: Our Silent Killer" for that half of our population that is either affected by or involved with this disease. This report is written in layman's language intended to be readily understood by anyone without special training in medicine. It includes a Glossary that carefully defines necessary medical terms. It includes an extensive list of scientific cites and references of the source data. It contains a Bibliography for further reading. It contains a Glycemic Index for those who daily face the consequences of faulty fat and carbohydrate metabolism but who still have to eat.

The report Insulin: Our Silent Killer provides an easily understood, workable alternative protocol for reversing this disease quickly, completely, permanently, naturally and economically without dangerous synthetic drugs in the vast majority of cases. This Special Report also discusses ways to reverse much of the collateral damage that unchecked proliferation of this disease causes in the body. This Special Report also provides effective non-drug therapies that are needed to control Blood Glucose during the time that the disease is being reversed but before our Automatic Blood Sugar Control System (BSCS) is again fully functional. All three elements of this approach are equally vital to curing this disease.

Insulin: Our Silent Killer relates the experiences I had when I was faced with this killer disease after I discovered that doctors treat but do not cure it. Most important, this Special Report contains a full detailed explanation of just where this disease comes from, what causes it, why it is not being routinely cured, a discussion of the economics of the disease, a discussion of the consequences of orthodox medical treatment, and of the reasons why the disease has become a national disgrace.

Our investigation and our report Insulin: Our Silent Killer has been featured at length in a very favorable book report in the spring, 1999 edition of Tom Valentine's "True Health" newsletter (Carotec, Inc., PO Box 9919, Naples, FL 34101 (941) 353-2348) and was given high marks by David Lawrence Dewey, nationally syndicated columnist, in his popular on line health advisory column:

and, we are currently the subject of favorable articles in other leading health oriented magazines. We are beginning to be involved with the exposing of this disgraceful epidemic throughout the media.

More recently, some of the research presented in this report was featured on the Good Morning America TV show as an investigative expose' of a major cause of this stealth disease epidemic. We are being very well received on nation wide talk radio everwhere we have been invited. This area of our work is expanding on its own momentum largely because of the real need for accurate information by those who are stricken by this disease.

For those who would like additional investigative leads we are preparing a links list to facilitate searching the world wide web.

To Order This Special Report

Within the US this report, Insulin:Our Silent Killer, can be ordered by sending $29.00 US, (we pay all postage and handling), together with your address to:

Thomas Smith
PO Box 7685
Loveland, CO 80537
If you are not a US resident Email us for the special shipping and payment instructions required for foreign transactions.
Or you can email us at:

Or you can call us at: 1 (970) 669-9176. Sorry, we are not set up to process credit card transactions.

This page is under construction as we search and locate other websites that contain useful additional information regarding the disease syndrome discussed at this site and/or related issues.. As we find and check out quality links we will post them on this page.

David Lawrence Dewey's column:

David is a best selling author and syndicated columnist that has a wealth of good information about the disease epidemic from which we all suffer. He often writes with wit and insight on health related issues the are of importance to us all.

Jan's Student nursing page:

This is a nursing power site. It about, for and by the nursing profession with numerous articles and discussions relative to the technology, art, challenges and issues of the medical business in general and the nursing profession in particular. An interesting and useful website maintained in a professional manner.

Shirley's wellness cafe

This is a large well researched site with an abundance of valuable health information on many topics. The material is well organized and presented in an easy to assimilate fashion. We link to topics of great interest to our study of this endocrine disorder with so many aliases. They are:

Oils that can save your life: Flax oil the wonder healer:
Diabetes: Alternative therapies:

Some additional website articles of interest:

And an excellent newsletter to which you may freely subscribe: